Gelişmiş Arama

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dc.contributor.authorIşık, Turgay
dc.contributor.authorAyhan, Erkan
dc.contributor.authorErgelen, Mehmet
dc.contributor.authorUyarel, Hüseyin
dc.date.accessioned2019-10-17T11:51:37Z
dc.date.available2019-10-17T11:51:37Z
dc.date.issued2012en_US
dc.identifier.issn0167-5273
dc.identifier.issn1874-1754
dc.identifier.urihttps://doi.org/10.1016/j.ijcard.2012.01.095
dc.identifier.urihttps://hdl.handle.net/20.500.12462/8788
dc.descriptionAyhan, Erkan (Balikesir Author)en_US
dc.description.abstractOxidative stress plays an important role in the development of endothelial dysfunction. The two important systems generating vascular oxidative stress are the nicotinamide adenine dinucleotide phosphate oxidase and the xanthine oxidase (XO) pathways. XO is an enzyme that produces uric acid (UA) during purine catabolism, and oxygen free radicals are generated [1]. Thus, the UA reflects level of oxidative stressen_US
dc.language.isoengen_US
dc.publisherElsevier Ireland Ltden_US
dc.relation.isversionof10.1016/j.ijcard.2012.01.095en_US
dc.rightsinfo:eu-repo/semantics/embargoedAccessen_US
dc.subjectCardiovascular Diseaseen_US
dc.subjectUric Aciden_US
dc.subjectRisk Factoren_US
dc.titleUric acid: A novel prognostic marker for cardiovascular diseaseen_US
dc.typeletteren_US
dc.relation.journalInternational Journal of Cardiologyen_US
dc.contributor.departmentTıp Fakültesien_US
dc.identifier.volume156en_US
dc.identifier.issue3en_US
dc.identifier.startpage328en_US
dc.identifier.endpage329en_US
dc.relation.publicationcategoryDiğeren_US


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